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Food intolerances

The 5 NutritolTM panels enables the identification of IgG-dependent food intolerances through the testing of 25 - 270 foods.

Balanced diet Dermatology Digestive disorders

What are the NutritolTM tests?

NutritolTM  tests several foods or food groups to detect possible food intolerances. They are available in three formats:

  • NutritolTM 25: Analysis of 25 common foods such as gluten, cow’s milk and certain fruit and vegetables.
  • NutritolTM 50: Analysis of 50 foods, the different varieties of meats, vegetables, cereals and dairy products.
  • NutritolTM 50 VEGAN : Analysis of 50 VEGAN foods, therefore only cereals, fruit and vegetables as well as spices.
  • NutritolTM 100 : Analysis of the 100 most important foods, large variety of dairy products, meat and fish, as well as fruit and vegetables.
  • NutritolTM 270: Analysis of 270 foods and additives: different varieties of meat, vegetables, fruit, cereals, dairy products, eggs, their alternatives and regional specialities as well as a large number of spices, tea, coffee, wine tannins, thickening agents and preservatives.

What can we expect from the results of NutritolTM assessments?

The IgG food intolerance test identifies the foods that cause disorders associated with these intolerances. As soon as you remove the IgG-positive foods that trigger these chronic inflammations, you reduce or eliminate your complaints.

Who is NutritolTM for?

Food intolerances can cause a wide variety of symptoms.That is why  NutritolTM  is particularly recommended for :

  • gastrointestinal symptoms,
  • non-coeliac gluten sensitivity,
  • chronic pain
  • weight issues,
  • psychological ailments,
  • skin problems,
  • migraines and headaches,
  • water retention,

More information on food intolerances panels

Method used

The method used by our laboratory for the determination of anti-food IgG antibodies is based on the principle of testing all IgG antibodies, namely IgG1 + IgG2 + IgG3 + IgG4.

The first three classes correspond to pro-inflammatory antibodies (as complement activators) and are potentially involved in the formation of immune complexes responsible for the deleterious effects of their presence; on the other hand, the IgG4 fraction does not have these properties and its presence corresponds rather to immune tolerance. Furthermore, IgG4 only represents 0.5% of the total IgG concentration: their level can therefore in no way distort the result of the total IgG assay, and it should also be remembered that tests measuring exclusively IgG4 are, for this reason, obviously to be rejected in the context of a food intolerance screening.

The number of positive reactions to food can, in addition to potentially revealing specific intolerances, help the diagnosis of IPH (intestinal hyperpermeability): more than 5 positive foods of different categories may raise the question of an alteration in the integrity of the intestinal epithelium, in which case, it may be advisable to explore or even treat by appropriate means, before considering the hypothesis of specific hypersensitivity.

The management of positive results is based on an exclusion protocol followed by reintroduction and finally stabilisation

For further reading...

Diet restriction in migraine, based on IgG against foods: a clinical double-blind, randomised, cross-over trial. Alpay K1, Ertas M, Orhan EK, Ustay DK, Lieners C, Baykan B. Cephalalgia. 2010 Jul;30(7):829-37.

IgG-based elimination diet in migraine plus irritable bowel syndrome. Aydinlar EI1, Dikmen PY, Tiftikci A, Saruc M, Aksu M, Gunsoy HG, Tozun N. Headache. 2013 Mar;53(3):514-25.

Clinical relevance of IgG antibodies against food antigens in Crohn’s disease: a double-blind cross-over diet intervention study. Bentz S1, Hausmann M, Piberger H, Kellermeier S, Paul S, Held L, Falk W, Obermeier F, Fried M, Schölmerich J, Rogler G. Digestion. 2010;81(4):252-64.

IgG Antibodies Against Food Antigens are Correlated with Inflammation and Intima Media Thickness in Obese Juveniles. M. Wilders-Truschnig, H. Mangge, C. Lieners, H-. J. Gruber, C. Mayer, W. März. Exp Clin Endocrinol Diabetes 2008; 116(4): 241-245.